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Location:  Home > Details for node YUC4
Type: Protein GRN ID: np02010 Name: YUC4
Annotation: YUC4 | Flavin-binding monooxygenase family protein [EC:1.14.13.168]
Gene Locus: AT5G11320
References:
  • Cheng Y, Dai X, Zhao Y. Auxin biosynthesis by the YUCCA flavin monooxygenases controls the formation of floral organs and vascular tissues in Arabidopsis. Genes Dev. 2006 Jul 1;20(13):1790-9. PubMed PMID: 16818609; PubMed Central PMCID: PMC1522075.
  • Cheng Y, Dai X, Zhao Y. Auxin synthesized by the YUCCA flavin monooxygenases is essential for embryogenesis and leaf formation in Arabidopsis. Plant Cell. 2007 Aug;19(8):2430-9. Epub 2007 Aug 17. PubMed PMID: 17704214; PubMed Central PMCID: PMC2002601.
  • Cheng Y, Qin G, Dai X, Zhao Y. NPY1, a BTB-NPH3-like protein, plays a critical role in auxin-regulated organogenesis in Arabidopsis. Proc Natl Acad Sci U S A. 2007 Nov 20;104(47):18825-9. Epub 2007 Nov 13. PubMed PMID: 18000043; PubMed Central PMCID: PMC2141861.
 
Pathways: Auxins 
Gene Expression (Affy ATH1 probeset): 250389_at    
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Relationship type:  protein modification     catalytic chemical reaction     protein-protein interaction    
compound-protein interaction     TF-target gene regulation     small RNA-target regulation    
co-expressed gene pair     carrier and transported molecule     family/complex and member
Relationship evidence:  Solid line: validated          Dashed line: predicted
Relationship direction: Arrow: positive          T shape: negative          None arrow: unknown or n/a
Hormone association: Thicker line: hormone related relationship         : hormone related Node
Node type:  Transcription factor or transcriptional regulator       Compound       Kinase
 Small regulatory RNA (miRNA & ta-siRNA)       Other nodes
No. Path
2 YUC4  --  is regulated by  --  SHI
3 YUC4  --  is regulated by  --  SRS1; STY1
4 YUC4  --  is a member of  --  YUC2 family19, coding gene
   Funding by the National Science Foundation    Funding by the Oklahoma Center for the Advancement of Science & Technology    Additional funding by the Samuel Roberts Noble Foundation

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